Because the ultimate goal of a steroid cycle is to increase strength and muscle size, the associated spike in estrogen which accompanies steroids such as Testosterone is considered undesirable. In order to disassociate the two effects, two classes of drug are used. Medications such as Nolvadex or Clomid target the estrogen receptors. They make it more difficult for the estrogen to exert it’s influence within the body thus allowing the testosterone to act more freely. The second class is aromatase inhibitors such as Femara. They target the aromatase enzyme itself in order to prevent the production of estrogen in the first place. Sometimes, it’s not always clear which option you should go with or even what the differences are between the two. Lets clear that up a little.
Information in regards to Turinabol was first published in 1962  and Chlorodehydromethyltestosterone was then manufactured and released by Jenapharm in East Germany as Turinabol and Oral Turinabol. Much like other compounds such as Anavar (Oxandrolone), Turinabol was revered by medical personnel and physicians for its ability to provide a fairly distinct separation of its anabolic and androgenic effects, favoring anabolic effects of course. This is one of the reasons why its often compared to compounds such as Anavar or Promobolan. As a result, much like Anavar or Primobolan , Oral-Turinabol saw extensive medical use in not just adult males but in women and children also. At the time, Turinabol was offered in two different concentrations per tablet: 1mg tablets, and 5mg tablets. The 1mg tablets would typically be utilized for individuals traditionally more sensitive to anabolic steroid therapies, such as women and children. Under its medical use at the time, it was prescribed for many different ailments, but was used frequently for the promotion of fat free mass in wasting disorders as well as the promotion of bone strength and mass.